Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A host parasite interaction rescues Drosophila oogenesis defects.

Nature - 4 Jul 2002

Starr Diana J, Cline Thomas W

Abstract excerpt

The cytoplasmically inherited bacterium Wolbachia pipientis is a widespread parasite of arthropods that manipulates the reproductive biology of its hosts, often to their detriment, in order to foster its own transmission through egg cytoplasm. Here we report that infection by Wolbachia restores fertility to Drosophila melanogaster mutant females prevented from making eggs by protein-coding lesions in Sex-lethal...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Genes, Lethal
Share to Host-Parasite Interactions
Share to Oogenesis

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to RNA-Binding Proteins

Share to Sex Characteristics

Share to Sex Determination Processes

Share to Wolbachia

Source

Identifiers

PMID: 12097909
DOI: 10.1038/nature00843

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A host parasite interaction rescues Drosophila oogenesis defects.PMID 12097909Read ↗

Effects of Wolbachia infection and ovarian tumor mutations on Sex-lethal germline functioning in Drosophila.Semantic proximity Read ↗

Wolbachia endosymbionts manipulate the self-renewal and differentiation of germline stem cells to reinforce fertility of their fruit fly hostSemantic proximity Read ↗

From Wolbachia genomics to phenotype: molecular models of cytoplasmic incompatibility must account for the multiplicity of compatibility types.Semantic proximity Read ↗

Induced paternal effects mimic cytoplasmic incompatibility in Drosophila.Semantic proximity Read ↗

Sperm chromatin remodelling and Wolbachia-induced cytoplasmic incompatibility in Drosophila.Semantic proximity Read ↗

A male-killing Wolbachia endosymbiont is concealed by another endosymbiont and a nuclear suppressor.Semantic proximity Read ↗

<i>Wolbachia</i> endosymbionts manipulate GSC self-renewal and differentiation to reinforce host fertilitySemantic proximity Read ↗

Cytoplasmic incompatibility and sperm cyst infection in different Drosophila-Wolbachia associations.Semantic proximity Read ↗

Loss of reproductive parasitism following transfer of male-killing Wolbachia to Drosophila melanogaster and Drosophila simulans.Semantic proximity Read ↗

Combined actions of bacteriophage-encoded genes in Wolbachia-induced male lethalitySemantic proximity Read ↗

A male-killing Wolbachia carries a feminizing factor and is associated with degradation of the sex-determining system of its host.Semantic proximity Read ↗

You can't keep a good parasite down: evolution of a male-killer suppressor uncovers cytoplasmic incompatibility.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.