Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Multi-omics insights of immune cells in the risk and prognosis of idiopathic membranous nephropathy

10 Aug 2025

Abstract excerpt

Idiopathic membranous nephropathy (IMN) is the major cause of autoimmune-related nephrotic syndrome. The role immune cells play in the risk and prognosis of IMN remains elusive. We employ multi-omics data and a variety of approaches to evaluate the causal link between 731 immune-cell phenotypes and IMN. In light of the findings emanating from Mendelian randomization analyses, only the regulatory T cell (Treg)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Renal Diseases and Glomerulopathies
Share to Systemic Lupus Erythematosus Research
Share to Vasculitis and related conditions

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 40783650
DOI: 10.1038/s42003-025-08642-3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Multi-omics insights of immune cells in the risk and prognosis of idiopathic membranous nephropathyPMID 40783650Read ↗

Combination scRNA-seq with GWAS identifies genetic risk factors in IgA nephropathy: An observational study.Semantic proximity Read ↗

Omics are Getting Us Closer to Understanding IgA NephropathySemantic proximity Read ↗

A Phenome-Wide Association and Mendelian Randomization Study of Genetically Predicted Membranous Nephropathy with Multiple Diseases.Semantic proximity Read ↗

Causal role of immune cells in diabetic nephropathy: a bidirectional Mendelian randomization study.Semantic proximity Read ↗

Mendelian randomization based on immune cells in diabetic nephropathy.Semantic proximity Read ↗

Membranous Nephropathy: Muti-omics Mendelian randomization and colocalization analysisSemantic proximity Read ↗

Revealing Potential Diagnostic Gene Biomarkers Associated with Immune Infiltration in Patients with Renal Fibrosis Based on Machine Learning AnalysisSemantic proximity Read ↗

Gene prediction of the causal relationship between immune cells and IgA nephropathy: A bidirectional Mendelian randomization study.Semantic proximity Read ↗

COVID-19 and membranous nephropathy: observational and Mendelian randomization analysesSemantic proximity Read ↗

The genetic architecture of membranous nephropathy and its potential to improve non-invasive diagnosis.Semantic proximity Read ↗

Causal role of immune cells in IgA nephropathy: a mendelian randomization study.Semantic proximity Read ↗

Immune cell signatures and inflammatory mediators: unraveling their genetic impact on chronic kidney disease through Mendelian randomization.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.